MALČÍKOVÁ, Jitka, Filip RÁZGA, Tomáš JURČEK, Dana DVOŘÁKOVÁ, Daniela ŽÁČKOVÁ, Hana TOŠKOVÁ, Ludmila ŠEBEJOVÁ, Jana ŠMARDOVÁ, Alexandra OLTOVÁ, Gabriela VAŇKOVÁ, Lenka JURAČKOVÁ, Martin TRBUŠEK, Šárka POSPÍŠILOVÁ, Jiří MAYER and Zdeněk RÁČIL. The BCR-ABL1 T315I mutation and additional genomic aberrations are dominant genetic lesions associated with disease progression in patients with chronic myelogenous leukemia resistant to tyrosine kinase inhibitor therapy. Leukemia & Lymphoma. London: Informa Healthcare, Gordon and Breach, 2013, vol. 54, No 9, p. 2083-2087. ISSN 1042-8194. Available from: https://dx.doi.org/10.3109/10428194.2012.762649.
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Basic information
Original name The BCR-ABL1 T315I mutation and additional genomic aberrations are dominant genetic lesions associated with disease progression in patients with chronic myelogenous leukemia resistant to tyrosine kinase inhibitor therapy.
Name in Czech Mutace BCR-ABL1 T315I a další genomové aberace dominantní genetické léze spojené s progresí onemocnění u pacientů s chronickou myeloidní leukémií odolné proti tyrosin kinázy léčbu inhibitorem.
Authors MALČÍKOVÁ, Jitka (203 Czech Republic, guarantor, belonging to the institution), Filip RÁZGA (703 Slovakia, belonging to the institution), Tomáš JURČEK (203 Czech Republic, belonging to the institution), Dana DVOŘÁKOVÁ (203 Czech Republic, belonging to the institution), Daniela ŽÁČKOVÁ (203 Czech Republic, belonging to the institution), Hana TOŠKOVÁ (203 Czech Republic, belonging to the institution), Ludmila ŠEBEJOVÁ (203 Czech Republic, belonging to the institution), Jana ŠMARDOVÁ (203 Czech Republic, belonging to the institution), Alexandra OLTOVÁ (203 Czech Republic, belonging to the institution), Gabriela VAŇKOVÁ (203 Czech Republic, belonging to the institution), Lenka JURAČKOVÁ (203 Czech Republic, belonging to the institution), Martin TRBUŠEK (203 Czech Republic, belonging to the institution), Šárka POSPÍŠILOVÁ (203 Czech Republic, belonging to the institution), Jiří MAYER (203 Czech Republic, belonging to the institution) and Zdeněk RÁČIL (203 Czech Republic, belonging to the institution).
Edition Leukemia & Lymphoma, London, Informa Healthcare, Gordon and Breach, 2013, 1042-8194.
Other information
Original language English
Type of outcome Article in a journal
Field of Study 30200 3.2 Clinical medicine
Country of publisher United Kingdom of Great Britain and Northern Ireland
Confidentiality degree is not subject to a state or trade secret
WWW URL
Impact factor Impact factor: 2.605
RIV identification code RIV/00216224:14740/13:00071791
Organization unit Central European Institute of Technology
Doi http://dx.doi.org/10.3109/10428194.2012.762649
UT WoS 000323492400044
Keywords in English BCR-ABL1; T315I; chronic myelogenous leukemia; therapy
Tags podil, rivok
Tags International impact, Reviewed
Changed by Changed by: Olga Křížová, učo 56639. Changed: 25/4/2014 20:11.
Abstract
The BCR-ABL1 T315I mutation and additional genomic aberrations are dominant genetic lesions associated with disease progression in patients with chronic myelogenous leukemia resistant to tyrosine kinase inhibitor therapy. The BCR-ABL1 T315I mutation and additional genomic aberrations are dominant genetic lesions associated with disease progression in patients with chronic myelogenous leukemia resistant to tyrosine kinase inhibitor therapy. The BCR-ABL1 T315I mutation and additional genomic aberrations are dominant genetic lesions associated with disease progression in patients with chronic myelogenous leukemia resistant to tyrosine kinase inhibitor therapy.
Abstract (in Czech)
Mutace BCR-ABL1 T315I a další genomové aberace dominantní genetické léze spojené s progresí onemocnění u pacientů s chronickou myeloidní leukémií odolné proti tyrosin kinázy léčbu inhibitorem. Mutace BCR-ABL1 T315I a další genomové aberace dominantní genetické léze spojené s progresí onemocnění u pacientů s chronickou myeloidní leukémií odolné proti tyrosin kinázy léčbu inhibitorem. Mutace BCR-ABL1 T315I a další genomové aberace dominantní genetické léze spojené s progresí onemocnění u pacientů s chronickou myeloidní leukémií odolné proti tyrosin kinázy léčbu inhibitorem.
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