J 2016

Prognostic Significance of Surfactant Protein A, Surfactant Protein D, Clara Cell Protein 16, S100 Protein, Trefoil Factor 3, and Prostatic Secretory Protein 94 in Idiopathic Pulmonary Fibrosis, Sarcoidosis, and Chronic Pulmonary Obstructive Disease

DOUBKOVÁ, Martina, Michal KARPISEK, Jiri MAZOCH, Jana SKŘIČKOVÁ, Michael DOUBEK et. al.

Basic information

Original name

Prognostic Significance of Surfactant Protein A, Surfactant Protein D, Clara Cell Protein 16, S100 Protein, Trefoil Factor 3, and Prostatic Secretory Protein 94 in Idiopathic Pulmonary Fibrosis, Sarcoidosis, and Chronic Pulmonary Obstructive Disease

Authors

DOUBKOVÁ, Martina (203 Czech Republic, belonging to the institution), Michal KARPISEK (203 Czech Republic), Jiri MAZOCH (203 Czech Republic), Jana SKŘIČKOVÁ (203 Czech Republic, guarantor, belonging to the institution) and Michael DOUBEK (203 Czech Republic, belonging to the institution)

Edition

Sarcoidosis Vasculitis and Diffuse Lung Diseases, Fidenza, Mattioli 1885, 2016, 1124-0490

Other information

Language

English

Type of outcome

Article in a journal

Field of Study

30203 Respiratory systems

Country of publisher

Italy

Confidentiality degree

is not subject to a state or trade secret

Impact factor

Impact factor: 1.575

RIV identification code

RIV/00216224:14110/16:00092064

Organization unit

Faculty of Medicine

UT WoS

000393274000005

Keywords in English

surfactant protein A; surfactant protein D; Clara cell protein 16; SIOO protein; trefoil factor 3

Tags

Tags

International impact, Reviewed
Changed: 22/3/2017 11:54, Ing. Mgr. Věra Pospíšilíková

Abstract

V originále

Background: Identification of serum and bronchoalveolar lavage fluid (BALF) biomarkers may facilitate diagnosis and prognostication in various lung disorders. Objective: Serum and BALF levels of surfactant protein A (SP-A), surfactant protein D (SP-D), Clara cell protein 16 (CC16), SIOO protein, trefoil factor 3 (TFF3), and prostatic secretory protein 94 (PSP94) were evaluated in 94 consecutive patients (idiopathic pulmonary fibrosis (IFF; n=18), sarcoidosis (n=25), chronic obstructive pulmonary disease (COPD; n=51)), and in 155 healthy controls. Methods: Biomarkers were measured at diagnosis and compared with disease characteristics. Both uniparametric and multiparametric analyses were used. Results: Seven significant correlations were found: 1) BALF PSP94 level correlated with prognosis of sarcoidosis (P=0.035); 2) BALF SP-D level with pulmonary functions in IFF (P=0.032); 3) BALF SP-D and TFF3 with IFF mortality (P=0.049 and 0.017, respectively); 4) serum TFF3 level with COPD mortality (P=0.006,); 5) serum SP-A with pulmonary functions impairment in IFF (P=0.011); 6) serum SP-D level was associated with HRCT interstitial score in IPF (P=0.0346); and 7) serum SP-A was associated with staging of COPD according to spirometry (P<0.001). Moreover, our analysis showed that some biomarker levels differed significantly among the diseases: 1) BALF SP-D level differed between sarcoidosis and IPF; 2) serum SP-A level differed among IPF, sarcoidosis, COPD and was also different from healthy controls; 3) serum S100A6, S100A11 levels differed among IPF, sarcoidosis, COPD from healthy controls 4) serum SP-D, CC16, TFF-3 levels distinguished IPF patients from healthy controls; and 5) serum CC16, TFF3, PSP94 distinguished COPD patients from healthy controls. Our study shows that some of selected biomarkers should have prognostic value in the analysed lung disorders. On the other hand, these biomarkers do not appear to be unequivocally suitable for differential diagnosis of these disorders.

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