Detailed Information on Publication Record
2017
Pupillary Disorders in Homonymous Visual Field Defects
SKORKOVSKÁ, Karolína, Barbara WILHELM and Helmut WILHELMBasic information
Original name
Pupillary Disorders in Homonymous Visual Field Defects
Authors
SKORKOVSKÁ, Karolína, Barbara WILHELM and Helmut WILHELM
Edition
1st. ed. Cham, Homonymous Visual Field Defects, p. 107-119, 13 pp. 2017
Publisher
Springer International Publishing
Other information
Language
English
Type of outcome
Kapitola resp. kapitoly v odborné knize
Field of Study
30207 Ophthalmology
Country of publisher
Switzerland
Confidentiality degree
není předmětem státního či obchodního tajemství
Publication form
printed version "print"
References:
Organization unit
Faculty of Medicine
ISBN
978-3-319-52282-1
Keywords in English
Pupil; Pupil light reflex ;Relative afferent pupillary defect; Pupil perimetry
Tags
Tags
International impact, Reviewed
Změněno: 26/2/2018 17:10, Soňa Böhmová
Abstract
V originále
Classically, the pupil light reflex pathway is considered to be a simple reflex arc consisting of the retinal ganglion cells, intercalated neurons in the midbrain, the oculomotor nerve, and short ciliary nerves. However, there are some specialties in the structure of the afferent pupillary pathway that should be taken into account when interpreting pupillary disorders and that can help in the topodiagnosis of the lesion. Moreover, studies in patients with lesions of the retrogeniculate pathway showed that the pupillary pathway is more complex than previously assumed and the retrogeniculate visual pathway and the visual cortex are also involved in the pupillary light reaction. Clear anatomic evidence is still lacking but pupillographic measurements in patients with various disorders of the visual pathway support the existence of two pupillomotor channels that drive the pupil light reaction – the subcortical (more primitive, luminance channel associated with the intrinsically photosensitive retinal ganglion cells) and the suprageniculate (responds to shifts in structured stimuli, is driven by the rods and cones, and receives input from the visual cortex and extrastriate areas). The chapter summarizes possible pupillary findings in patients with homonymous hemianopia.