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@inproceedings{712472, author = {Pásek, Michal and Bébarová, Markéta and Matejovič, Peter and Nováková, Marie}, address = {Brno}, booktitle = {Human Biomechanics 2006}, keywords = {IKto-channel;haloperidol;quantitative model}, language = {eng}, location = {Brno}, isbn = {80-214-3232-2}, pages = {1-5}, publisher = {University of Technology}, title = {Interaction of haloperidol with IKto-channels in cardiac cells: a quantitative model}, year = {2006} }
TY - JOUR ID - 712472 AU - Pásek, Michal - Bébarová, Markéta - Matejovič, Peter - Nováková, Marie PY - 2006 TI - Interaction of haloperidol with IKto-channels in cardiac cells: a quantitative model PB - University of Technology CY - Brno SN - 8021432322 KW - IKto-channel;haloperidol;quantitative model N2 - Haloperidol inhibited the transient outward potassium current IKto with decrease of peak current, acceleration of the time course of apparent inactivation and deceleration of recovery from inactivation and block. The quantitative model predicted interaction of haloperidol with both open and open-inactivated states as the most probable mechanism of IKto-block. ER -
PÁSEK, Michal, Markéta BÉBAROVÁ, Peter MATEJOVIČ and Marie NOVÁKOVÁ. Interaction of haloperidol with IKto-channels in cardiac cells: a quantitative model. In \textit{Human Biomechanics 2006}. Brno: University of Technology, 2006, p.~1-5. ISBN~80-214-3232-2.
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