C 2017

Pupillary Disorders in Homonymous Visual Field Defects

SKORKOVSKÁ, Karolína, Barbara WILHELM a Helmut WILHELM

Základní údaje

Originální název

Pupillary Disorders in Homonymous Visual Field Defects

Autoři

SKORKOVSKÁ, Karolína, Barbara WILHELM a Helmut WILHELM

Vydání

1st. ed. Cham, Homonymous Visual Field Defects, od s. 107-119, 13 s. 2017

Nakladatel

Springer International Publishing

Další údaje

Jazyk

angličtina

Typ výsledku

Kapitola resp. kapitoly v odborné knize

Obor

30207 Ophthalmology

Stát vydavatele

Švýcarsko

Utajení

není předmětem státního či obchodního tajemství

Forma vydání

tištěná verze "print"

Odkazy

Organizační jednotka

Lékařská fakulta

ISBN

978-3-319-52282-1

Klíčová slova anglicky

Pupil; Pupil light reflex ;Relative afferent pupillary defect; Pupil perimetry

Štítky

Příznaky

Mezinárodní význam, Recenzováno
Změněno: 26. 2. 2018 17:10, Soňa Böhmová

Anotace

V originále

Classically, the pupil light reflex pathway is considered to be a simple reflex arc consisting of the retinal ganglion cells, intercalated neurons in the midbrain, the oculomotor nerve, and short ciliary nerves. However, there are some specialties in the structure of the afferent pupillary pathway that should be taken into account when interpreting pupillary disorders and that can help in the topodiagnosis of the lesion. Moreover, studies in patients with lesions of the retrogeniculate pathway showed that the pupillary pathway is more complex than previously assumed and the retrogeniculate visual pathway and the visual cortex are also involved in the pupillary light reaction. Clear anatomic evidence is still lacking but pupillographic measurements in patients with various disorders of the visual pathway support the existence of two pupillomotor channels that drive the pupil light reaction – the subcortical (more primitive, luminance channel associated with the intrinsically photosensitive retinal ganglion cells) and the suprageniculate (responds to shifts in structured stimuli, is driven by the rods and cones, and receives input from the visual cortex and extrastriate areas). The chapter summarizes possible pupillary findings in patients with homonymous hemianopia.