Higher rates of molecular response to Peg-IFNa in JAK2V617F vs. Calr mutant MPN patients are due to JAK1-mediated STAT1 activation and autoregulation.

CZECH, J., S. CORDUA, Barbora WEINBERGEROVÁ, L. HAN, C. SCHUBERT, T. BRÜMMENDORF, Jiří MAYER, Zdeněk RÁČIL, Blanka KUBEŠOVÁ, TA. KNUDSEN, A. SORENSEN, M. HOLMSTROM, L. KJAER, V. SKOV, T. STAUFFER LARSEN, HC. HASSELBALCH, N. CHATAIN and S. KOSCHMIEDER. Higher rates of molecular response to Peg-IFNa in JAK2V617F vs. Calr mutant MPN patients are due to JAK1-mediated STAT1 activation and autoregulation. In 59th ASH Annual Meeting and Exposition, USA, Atlanta. 2017.

Basic information

Original name

Higher rates of molecular response to Peg-IFNa in JAK2V617F vs. Calr mutant MPN patients are due to JAK1-mediated STAT1 activation and autoregulation.

Authors

CZECH, J., S. CORDUA, Barbora WEINBERGEROVÁ, L. HAN, C. SCHUBERT, T. BRÜMMENDORF, Jiří MAYER, Zdeněk RÁČIL, Blanka KUBEŠOVÁ, TA. KNUDSEN, A. SORENSEN, M. HOLMSTROM, L. KJAER, V. SKOV, T. STAUFFER LARSEN, HC. HASSELBALCH, N. CHATAIN and S. KOSCHMIEDER

Edition

59th ASH Annual Meeting and Exposition, USA, Atlanta, 2017

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Other information

Type of outcome

Konferenční abstrakt

Confidentiality degree

není předmětem státního či obchodního tajemství

Organization unit

Faculty of Medicine