Cerebellum and dystonia: The story continues. Will the patients benefit from new discoveries?

BAREŠ, Martin and Pavel FILIP. Cerebellum and dystonia: The story continues. Will the patients benefit from new discoveries? Clinical Neurophysiology. Clare: Elsevier Ireland, 2018, vol. 129, No 1, p. 282-283. ISSN 1388-2457. Available from: https://dx.doi.org/10.1016/j.clinph.2017.10.007.

Basic information

• Original name: Cerebellum and dystonia: The story continues. Will the patients benefit from new discoveries?
• Authors: BAREŠ, Martin (203 Czech Republic, guarantor, belonging to the institution) and Pavel FILIP (703 Slovakia, belonging to the institution).
• Edition: Clinical Neurophysiology, Clare, Elsevier Ireland, 2018, 1388-2457.

Other information

• Original language: English
• Type of outcome: Article in a journal
• Field of Study: 30210 Clinical neurology
• Country of publisher: Ireland
• Confidentiality degree: is not subject to a state or trade secret
• WWW: URL
• Impact factor: Impact factor: 3.675
• RIV identification code: RIV/00216224:14110/18:00102800
• Organization unit: Faculty of Medicine
• Doi: http://dx.doi.org/10.1016/j.clinph.2017.10.007
• UT WoS: 000418602000034
• Keywords in English: Cerebellum; dystonia
• Tags: rivok
• Tags: International impact, Reviewed

Abstract

When reading the recent paper of Avanzino and colleagues with cogent evidence supporting a possible role of the cerebellum in the expression of the clinical phenotype in dystonia (Avanzino et al., 2018, this issue of Clinical Neurophysiology), I could not resist to feel reminded of an invited review paper published in 2013 in the Clinical Neurophysiology, where we provided a state-of-the-art overview of cerebellar involvement in dystonia, even with possible therapeutic targets for dystonic patients in the future (Filip et al., 2017). The last couple of years have offered to me a lot of discussions on this emerging issue, positive, negative as well as neutral, during scientific sessions organized both nationally and internationally, as the traditional view of the pathophysiology of dystonia being linked mainly to basal ganglia was challenged more and more.