HALUZOVÁ, Adéla, René JURA, Josef BEDNAŘÍK, Světlana SKUTILOVÁ, Tereza ANDRAŠINOVÁ, Miloš KEŘKOVSKÝ and Andrea KŘIVANOVÁ. Subakutně probíhající reverzibilní hypertenzní leukoencefalopatie - kazuistika (Subacute Hypertensive Reversible Leukoencephalopathy - a Case Report). CESKA A SLOVENSKA NEUROLOGIE A NEUROCHIRURGIE. Praha: Česká lékařská společnost J.E.Purkyně, 2008, vol. 71, No 6, p. 726-731. ISSN 1210-7859.
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Basic information
Original name Subakutně probíhající reverzibilní hypertenzní leukoencefalopatie - kazuistika
Name (in English) Subacute Hypertensive Reversible Leukoencephalopathy - a Case Report
Authors HALUZOVÁ, Adéla (203 Czech Republic, guarantor, belonging to the institution), René JURA (203 Czech Republic, belonging to the institution), Josef BEDNAŘÍK (203 Czech Republic, belonging to the institution), Světlana SKUTILOVÁ (203 Czech Republic, belonging to the institution), Tereza ANDRAŠINOVÁ (203 Czech Republic, belonging to the institution), Miloš KEŘKOVSKÝ (203 Czech Republic, belonging to the institution) and Andrea KŘIVANOVÁ (203 Czech Republic, belonging to the institution).
Edition CESKA A SLOVENSKA NEUROLOGIE A NEUROCHIRURGIE, Praha, Česká lékařská společnost J.E.Purkyně, 2008, 1210-7859.
Other information
Original language Czech
Type of outcome Article in a journal
Field of Study 30103 Neurosciences
Country of publisher Czech Republic
Confidentiality degree is not subject to a state or trade secret
Impact factor Impact factor: 0.319
RIV identification code RIV/00216224:14110/08:00107022
Organization unit Faculty of Medicine
UT WoS 000261867200013
Keywords in English hypertensive encephalopathy; leukoencephalopathy
Tags Reviewed
Changed by Changed by: Mgr. Michal Petr, učo 65024. Changed: 30/4/2020 00:41.
Abstract
Hypertensive encephalopathy induced by essential or secondary arterial hypertension is characterised by a sudden onset of symptoms pointing to diffuse encephalopathy (headache, nausea, vomiting, vision problems, confusion or epileptic seizures) linked with an increase in blood pressure (BP, hypertensive crisis). in such case, the imaging methods applied, especially magnetic resonance (MR), show leukoencephalopathy. Both clinical symptoms and MR imaging findings are reversible after the blood pressure values have been normalised. We report a case of a 37-year old man who developed non-specific symptoms of encephalopathy over the period of one year (cephalea, vision disorders, behaviour changes, atactic walk). CT and MRI of the patient's brain showed diffuse affection of the white matter of the brain, cerebellum and of the cerebral trunk. Inflammatory, demyelinisating, ischaemic, oncologic, metabolic and toxic aetiologies of leukoencephalopathy were excluded. In spite of absence of anamnestic data on arterial hypertension, and of normotensive values at admission, a hypertension crisis developed and clinical symptoms progressed into a picture of delirium. Blood pressure compensation resulted in the regression of clinical symptoms and a significant attenuation of cardiologic signs of encephalopathy, which was a confirmation of hypertensive etiology of leukoencephalopathy. Subsequently, the presence of chronic idiopathic arterial hypertension was confirmed, and secondary causes of hypertension including phaeochromocytoma were excluded. Hypertensive encephalopathy should be considered also in the case of protracted and fluctuating symptoms of encephalopathy and normal results for current blood pressure measurements.
Abstract (in English)
Hypertensive encephalopathy induced by essential or secondary arterial hypertension is characterised by a sudden onset of symptoms pointing to diffuse encephalopathy (headache, nausea, vomiting, vision problems, confusion or epileptic seizures) linked with an increase in blood pressure (BP, hypertensive crisis). in such case, the imaging methods applied, especially magnetic resonance (MR), show leukoencephalopathy. Both clinical symptoms and MR imaging findings are reversible after the blood pressure values have been normalised. We report a case of a 37-year old man who developed non-specific symptoms of encephalopathy over the period of one year (cephalea, vision disorders, behaviour changes, atactic walk). CT and MRI of the patient's brain showed diffuse affection of the white matter of the brain, cerebellum and of the cerebral trunk. Inflammatory, demyelinisating, ischaemic, oncologic, metabolic and toxic aetiologies of leukoencephalopathy were excluded. In spite of absence of anamnestic data on arterial hypertension, and of normotensive values at admission, a hypertension crisis developed and clinical symptoms progressed into a picture of delirium. Blood pressure compensation resulted in the regression of clinical symptoms and a significant attenuation of cardiologic signs of encephalopathy, which was a confirmation of hypertensive etiology of leukoencephalopathy. Subsequently, the presence of chronic idiopathic arterial hypertension was confirmed, and secondary causes of hypertension including phaeochromocytoma were excluded. Hypertensive encephalopathy should be considered also in the case of protracted and fluctuating symptoms of encephalopathy and normal results for current blood pressure measurements.
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