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@article{828354, author = {Chlebová, Katarína and Bryja, Vítězslav and Dvořák, Petr and Kozubík, Alois and Wilcox, W.R. and Krejčí, Pavel}, article_location = {Switzerland}, article_number = {2}, doi = {http://dx.doi.org/10.1007/s00018-008-8440-4}, keywords = {FGF2; fibroblast growth factor;nuclear; signalling; Api5; SMN}, language = {eng}, issn = {1420-682X}, journal = {Cellular and molecular life sciences : CMLS}, title = {High molecular weight FGF2: the biology of a nuclear growth factor}, volume = {66}, year = {2009} }
TY - JOUR ID - 828354 AU - Chlebová, Katarína - Bryja, Vítězslav - Dvořák, Petr - Kozubík, Alois - Wilcox, W.R. - Krejčí, Pavel PY - 2009 TI - High molecular weight FGF2: the biology of a nuclear growth factor JF - Cellular and molecular life sciences : CMLS VL - 66 IS - 2 SP - 225-235 EP - 225-235 SN - 1420682X KW - FGF2 KW - fibroblast growth factor;nuclear KW - signalling KW - Api5 KW - SMN N2 - Fibroblast growth factor 2 (FGF2) is one of the most studied growth factors to date. Most attention has been dedicated to the smallest, 18 kDa FGF2 variant that is released by cells and acts through activation of cell-surface FGF-receptor tyrosine kinases. There are, however, several higher molecular weight (HMW) variants of FGF2 that rarely leave their producing cells, are retained in the nucleus and act independently of FGF-receptors (FGFR). Despite significant evidence documenting the expression and intracellular trafficking of HMW FGF2, many important questions remain about the physiological roles and mechanisms of action of HMW FGF2. In this review, we summarize the current knowledge about the biology of HMW FGF2, its role in disease and areas for future investigation. ER -
CHLEBOVÁ, Katarína, Vítězslav BRYJA, Petr DVOŘÁK, Alois KOZUBÍK, W.R. WILCOX a Pavel KREJČÍ. High molecular weight FGF2: the biology of a nuclear growth factor. \textit{Cellular and molecular life sciences : CMLS}. Switzerland, 2009, roč.~66, č.~2, s.~225-235. ISSN~1420-682X. Dostupné z: https://dx.doi.org/10.1007/s00018-008-8440-4.
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