J 2020

Out-of-Hospital Cardiac Arrest Due to Ventricular Fibrillation in a 5-Year-Old Pediatric Patient

KLUČKA, Jozef; Tomáš JUŘENČÁK; Petr ŠTOURAČ; Pavel VÍT; Vladimíra FORALOVÁ et al.

Základní údaje

Originální název

Out-of-Hospital Cardiac Arrest Due to Ventricular Fibrillation in a 5-Year-Old Pediatric Patient

Autoři

KLUČKA, Jozef; Tomáš JUŘENČÁK; Petr ŠTOURAČ; Pavel VÍT; Vladimíra FORALOVÁ a Iva SYNKOVÁ

Vydání

PEDIATRIC EMERGENCY CARE, PHILADELPHIA, LIPPINCOTT WILLIAMS & WILKINS, 2020, 0749-5161

Další údaje

Jazyk

angličtina

Typ výsledku

Článek v odborném periodiku

Obor

30209 Paediatrics

Stát vydavatele

Spojené státy

Utajení

není předmětem státního či obchodního tajemství

Odkazy

Impakt faktor

Impact factor: 1.454

Označené pro přenos do RIV

Ano

Kód RIV

RIV/00216224:14110/20:00116127

Organizační jednotka

Lékařská fakulta

EID Scopus

Klíčová slova anglicky

ventricular fibrillation; cardiac arrest; postresuscitation care

Příznaky

Mezinárodní význam, Recenzováno
Změněno: 5. 8. 2020 07:56, Mgr. Tereza Miškechová

Anotace

V originále

Out-of-hospital cardiac arrest in pediatric population is rare and predominantly has respiratory aetiology. Authors present the relatively unique case of out-of hospital cardiac arrest in 5-years old pediatric patient due to ventricular fibrillation (VF) as the initial rhythm during the advanced life support. The patient was resuscitated by his parents and the initial rhythm was VF. After defibrillation the patient was admitted to the pediatric intensive care were another two episodes of VF was detected and treated. After standard postresuscitation care, patient was weaned from sedation and extubated with good neurologic outcome. Genetic screening of the 7 genes associated with cardiac channelopathies (KCNQ1, KCNH2, SCN5A, KCNE1, KCNE2, RYR2, CASQ2) found mutation in gene KCHN2 and gene SCN5A, that were according to actual data considered benign. This case highlights the need for automated external defibrillator implementation in basic life support also in pediatric population and possible role of genetic predisposition in emergence of fibrillation.