J 2025

Deleterious variants in the autophagy-related gene RB1CC1/FIP200 impair immunity to SARS-CoV-2

HU, Lili; Renee M. VAN DER SLUIS; Kennith Brian CASTELINO; Bao-Cun ZHANG; Andreas RONIT et al.

Základní údaje

Originální název

Deleterious variants in the autophagy-related gene RB1CC1/FIP200 impair immunity to SARS-CoV-2

Autoři

HU, Lili; Renee M. VAN DER SLUIS; Kennith Brian CASTELINO; Bao-Cun ZHANG; Andreas RONIT; Thomas ZILLINGER; Marvin WERNER; Sofie Eg JORGENSEN; Anne Louise HANSEN; Alice PEDERSEN; Ryo NARITA; Line S. REINERT; Bettina BUNDGAARD; Laurent ABEL; Alessandro AIUTI; Saleh AL-MUHSEN; Fahd AL-MULLA; Mark S. ANDERSON; Evangelos ANDREAKOS; Andres A. ARIAS; Lisa M. ARKIN; Hagit Baris FELDMAN; Paul BASTARD; Alexandre BELOT; Catherine M. BIGGS; Dusan BOGUNOVIC; Alexandre BOLZE; Anastasiia BONDARENKO; Alessandro BORGHESI; Ahmed A. BOUSFIHA; Petter BRODIN; Yenan BRYCESON; Giorgio CASARI; John CHRISTODOULOU; Aurelie COBAT; Roger COLOBRAN; Antonio CONDINO-NETO; Stefan N. CONSTANTINESCU; Megan A. COOPER; Clifton L. DALGARD; Murkesh DESAI; Beth A. DROLET; Munis DUNDAR; Xavier DUVAL; Sotirija DUVLIS; Jamila EL BAGHDADI; Philippine ELOY; Sara ESPINOSA-PADILLA; Jacques FELLAY; Carlos FLORES; Jose Luis FRANCO; Antoine FROIDURE; Guy GOROCHOV; Marta GUT; Filomeen HAERYNCK; David HAGIN; Rabih HALWANI; Lennart HAMMARSTROM; James R. HEATH; Elena W. Y. HSIEH; Eystein HUSEBYE; Kohsuke IMAI; Yuval ITAN; Emmanuelle JOUANGUY; Elzbieta KAJA; Timokratis KARAMITROS; Kai KISAND; Cheng-Lung KU; Yu-Lung LAU; Ling YUN; Carrie L. LUCAS; Matthieu MAHEVAS; Davood MANSOURI; Laszlo MARODI; France MENTRE; Isabelle MEYTS; Joshua D. MILNER; Kristina MIRONSKA; Tomohiro MORIO; Lisa F. P. NG; Luigi D. NOTARANGELO; Antonio NOVELLI; Giuseppe NOVELLI; Cliona O'FARRELLY; Satoshi OKADA; Keisuke OKAMOTO; Tayfun OZCELIK; Firat OZCELIK; Qiang PAN-HAMMARSTROM; Rebeca DE DIEGO PEREZ; Jordi PEREZ-TUR; David S. PERLIN; Jonny PETER; Anna M. PLANAS; Carolina PRANDO; Aurora PUJOL; Anne PUEL; Lluis QUINTANA-MURCI; Sathishkumar RAMASWAMY; Laurent RENIA; Igor RESNICK; Carlos RODRIGUEZ-GALLEGO; Vanessa SANCHO-SHIMIZU; Anna SEDIVA; Mikko R. J. SEPPANEN; Mohammad SHAHROOEI; Anna SHCHERBINA; Ondřej SLABÝ; Andrew L. SNOW; Pere SOLER-PALACIN; Vassili SOUMELIS; Andras N. SPAAN; Helen C. SU; Ivan TANCEVSKI; Stuart G. TANGYE; Ahmad Abou TAYOUN; Sehime Gulsun TEMEL; Christian THORBALL; Pierre TIBERGHIEN; Sophie TROUILLET-ASSANT; Stuart E. TURVEY; K. M. Furkan UDDIN; Mohammed J. UDDIN; Diederik VAN DE BEEK; Fernanda Sales Luiz VIANNA; Donald C. VINH; Horst VON BERNUTH; Joost WAUTERS; Mayana ZATZ; Qian ZHANG; Shen-Ying ZHANG; Jacob BODILSEN; Kristoffer Skaalum HANSEN; Merete STORGAARD; Thomas BENFIELD; Marie HELLEBERG; Christian K. HOLM; Aurelie COBAT; Jean-Laurent CASANOVA; Fulvio REGGIORI; Muriel MARI; Soren R. PALUDAN a Trine H. MOGENSEN

Vydání

Nature communications, BERLIN, NATURE PORTFOLIO, 2025, 2041-1723

Další údaje

Jazyk

angličtina

Typ výsledku

Článek v odborném periodiku

Obor

10606 Microbiology

Stát vydavatele

Spojené státy

Utajení

není předmětem státního či obchodního tajemství

Odkazy

Impakt faktor

Impact factor: 15.700 v roce 2024

Označené pro přenos do RIV

Ano

Kód RIV

RIV/00216224:14110/25:00143096

Organizační jednotka

Lékařská fakulta

EID Scopus

Klíčová slova anglicky

ENDOPLASMIC-RETICULUM TURNOVER; INDEPENDENT FUNCTIONS; VIRUS-INFECTION; RECEPTOR; PROTEINS; PATHWAY

Štítky

Příznaky

Mezinárodní význam, Recenzováno
Změněno: 26. 2. 2026 13:09, Mgr. Eva Dubská

Anotace

V originále

The clinical outcome of SARS-CoV-2 infection spans from asymptomatic viral elimination to lethal COVID-19 pneumonia, which is due to type I interferon (IFN) deficiency in at least 15-20% of cases. We report two unrelated male patients with critical COVID-19 who are heterozygous for rare deleterious variants in RB1CC1, encoding the autophagy-related FIP200 protein. Airway epithelial cells genetically deprived of FIP200 or cell lines expressing the RB1CC1/FIP200 patient variants exhibit elevated SARS-CoV-2 replication and impaired autophagic flux. The antiviral function of FIP200 is independent of canonical autophagy and type I IFN, but involves the selective autophagy receptor NDP52. We identify a non-canonical function of FIP200 in a novel lysosomal degradation pathway, in which SARS-CoV-2 virions are targeted to single-membrane compartments for degradation of viral RNA in LC3B-positive acidified vesicles. This pathway is impaired in FIP200-deficient cells and in cells expressing FIP200 patient haplotypes. Collectively, we describe a cell-autonomous anti-SARS-CoV-2 restriction pathway, dependent on FIP200 and NDP52, and independent of canonical autophagy and type I IFN, which can underlie critical COVID-19 pneumonia.