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@inproceedings{712472,
author = {Pásek, Michal and Bébarová, Markéta and Matejovič, Peter and Nováková, Marie},
address = {Brno},
booktitle = {Human Biomechanics 2006},
keywords = {IKto-channel;haloperidol;quantitative model},
language = {eng},
location = {Brno},
isbn = {80-214-3232-2},
pages = {1-5},
publisher = {University of Technology},
title = {Interaction of haloperidol with IKto-channels in cardiac cells: a quantitative model},
year = {2006}
}
TY - JOUR
ID - 712472
AU - Pásek, Michal - Bébarová, Markéta - Matejovič, Peter - Nováková, Marie
PY - 2006
TI - Interaction of haloperidol with IKto-channels in cardiac cells: a quantitative model
PB - University of Technology
CY - Brno
SN - 8021432322
KW - IKto-channel;haloperidol;quantitative model
N2 - Haloperidol inhibited the transient outward potassium current IKto with decrease of peak current, acceleration of the time course of apparent inactivation and deceleration of recovery from inactivation and block. The quantitative model predicted interaction of haloperidol with both open and open-inactivated states as the most probable mechanism of IKto-block.
ER -
PÁSEK, Michal, Markéta BÉBAROVÁ, Peter MATEJOVIČ a Marie NOVÁKOVÁ. Interaction of haloperidol with IKto-channels in cardiac cells: a quantitative model. In \textit{Human Biomechanics 2006}. Brno: University of Technology, 2006, s.~1-5. ISBN~80-214-3232-2.
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