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@article{984074,
author = {Rédová, Martina and Svoboda, Marek and Slabý, Ondřej},
article_location = {San Diego},
article_number = {2},
doi = {http://dx.doi.org/10.1016/j.bbrc.2011.01.019},
keywords = {MicroRNA; Renal cell carcinoma; Hypoxia; Epithelial-mesenchymal transition; Proliferation; Apoptosis},
language = {eng},
issn = {0006-291X},
journal = {Biochem Biophys Res Commun},
title = {MicroRNAs and their target gene networks in renal cell carcinoma},
url = {http://www.sciencedirect.com/science/article/pii/S0006291X11000386},
volume = {405},
year = {2011}
}
TY - JOUR
ID - 984074
AU - Rédová, Martina - Svoboda, Marek - Slabý, Ondřej
PY - 2011
TI - MicroRNAs and their target gene networks in renal cell carcinoma
JF - Biochem Biophys Res Commun
VL - 405
IS - 2
SP - 153-156
EP - 153-156
PB - Elsevier Science
SN - 0006291X
KW - MicroRNA
KW - Renal cell carcinoma
KW - Hypoxia
KW - Epithelial-mesenchymal transition
KW - Proliferation
KW - Apoptosis
UR - http://www.sciencedirect.com/science/article/pii/S0006291X11000386
L2 - http://www.sciencedirect.com/science/article/pii/S0006291X11000386
N2 - MiRNAs are related to the processes of cell proliferation, apoptosis, angiogenesis, invasion, and metastasis in RCC. MiRNAs expression profiles are associated with several RCC-specific genetic alterations. It has been well documented that several miRNAs are downstream effector molecules of the HIF-induced hypoxia response. MiR-200 family is linked to epithelial-mesenchymal transition which is one of the most significant pathogenetic mechanism in RCC. Mechanistic studies in RCC have provided the rationale of using miRNAs as potential therapeutic targets.
ER -
RÉDOVÁ, Martina, Marek SVOBODA a Ondřej SLABÝ. MicroRNAs and their target gene networks in renal cell carcinoma. \textit{Biochem Biophys Res Commun}. San Diego: Elsevier Science, 2011, roč.~405, č.~2, s.~153-156. ISSN~0006-291X. Dostupné z: https://dx.doi.org/10.1016/j.bbrc.2011.01.019.
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